Jun Ma

Publication Activity (10 Years)

Years Active: 2023-2024
Publications (10 Years): 9

Top Topics

Traumatic Brain Injury

Subarachnoid Hemorrhage

Histone Deacetylase

Top Venues

CNS neuroscience & therapeutics

This page only lists publications with an associated author ORCID identifier.

Publications

Haoming Shu, Xin Zhang, Yingyan Pu, Yinuo Zhang, Shixue Huang, Jun Ma, Li Cao, Xuhui Zhou
Fucoidan improving spinal cord injury recovery: Modulating microenvironment and promoting remyelination. CNS neuroscience & therapeutics 30 (8) (2024)
Haoming Shu, Xin Zhang, Yingyan Pu, Yinuo Zhang, Shixue Huang, Jun Ma, Li Cao, Xuhui Zhou
Fucoidan improving spinal cord injury recovery: Modulating microenvironment and promoting remyelination. CNS neuroscience & therapeutics 30 (8) (2024)
Qingjie Kong, Fudong Li, Kaiqiang Sun, Xiaofei Sun, Jun Ma
Valproic acid ameliorates cauda equina injury by suppressing HDAC2-mediated ferroptosis. CNS neuroscience & therapeutics 30 (4) (2023)
Qingjie Kong, Fudong Li, Kaiqiang Sun, Xiaofei Sun, Jun Ma
Valproic acid ameliorates cauda equina injury by suppressing HDAC2-mediated ferroptosis. CNS neuroscience & therapeutics 30 (4) (2023)
Qingjie Kong, Fudong Li, Kaiqiang Sun, Xiaofei Sun, Jun Ma
Valproic acid ameliorates cauda equina injury by suppressing HDAC2-mediated ferroptosis. CNS neuroscience & therapeutics 30 (4) (2023)
Qingjie Kong, Fudong Li, Kaiqiang Sun, Xiaofei Sun, Jun Ma
Valproic acid ameliorates cauda equina injury by suppressing HDAC2-mediated ferroptosis. CNS neuroscience & therapeutics 30 (4) (2023)
Qingjie Kong, Fudong Li, Kaiqiang Sun, Xiaofei Sun, Jun Ma
Valproic acid ameliorates cauda equina injury by suppressing HDAC2-mediated ferroptosis. CNS neuroscience & therapeutics 30 (4) (2023)
Qingjie Kong, Fudong Li, Kaiqiang Sun, Xiaofei Sun, Jun Ma
Valproic acid ameliorates cauda equina injury by suppressing HDAC2-mediated ferroptosis. CNS neuroscience & therapeutics (2023)
Qingjie Kong, Fudong Li, Kaiqiang Sun, Xiaofei Sun, Jun Ma
Valproic acid ameliorates cauda equina injury by suppressing HDAC2-mediated ferroptosis. CNS neuroscience & therapeutics 30 (4) (2023)