Kenn Gerdes

ResearcherID ORCID

Publication Activity (10 Years)

Years Active: 2017-2020
Publications (10 Years): 9

Top Venues

Molecular microbiology

mBio

Science signaling

MicrobiologyOpen

Technical University of Denmark (PHD)

This page only lists publications with an associated author ORCID identifier.

Education History

Technical University of Denmark Sep 1o 1983 - Aug 30o 1986

Denmark

PHD :: Department of Microbiology

Publications

Cinzia Fino, Martin Vestergaard, Hanne Ingmer, Fabien Pierrel, Kenn Gerdes, Alexander Harms
PasT of Escherichia coli sustains antibiotic tolerance and aerobic respiration as a bacterial homolog of mitochondrial Coq10. MicrobiologyOpen 9 (8) (2020)
Anurag Kumar Sinha, Kristoffer Skovbo Winther, Mohammad Roghanian, Kenn Gerdes
Fatty acid starvation activates RelA by depleting lysine precursor pyruvate. Molecular microbiology 112 (4) (2019)
Stine Vang Nielsen, Kathryn Jane Turnbull, Mohammad Roghanian, Rene Bærentsen, Maja Semanjski, Ditlev E Brodersen, Boris Macek, Kenn Gerdes
Serine-Threonine Kinases Encoded by Split hipA Homologs Inhibit Tryptophanyl-tRNA Synthetase. mBio 10 (3) (2019)
Ragnhild Bager Skjerning, Meriem Senissar, Kristoffer S Winther, Kenn Gerdes, Ditlev E Brodersen
The RES domain toxins of RES-Xre toxin-antitoxin modules induce cell stasis by degrading NAD+ Molecular microbiology 111 (1) (2018)
Maja Semanjski, Elsa Germain, Katrin Bratl, Andreas R Kiessling, Kenn Gerdes, Boris Macek
The kinases HipA and HipA7 phosphorylate different substrate pools in Escherichia coli to promote multidrug tolerance. Science signaling 11 (547) (2018)
Yong Zhang, Eva Zborníková, Dominik Rejman, Kenn Gerdes
Novel (p)ppGpp Binding and Metabolizing Proteins of Escherichia coli. mBio 9 (2) (2018)
Julia Piskunova, Etienne Maisonneuve, Elsa Germain, Kenn Gerdes, Konstantin V Severinov
Peptide-nucleotide antibiotic Microcin C is a potent inducer of stringent response and persistence in both sensitive and producing cells. Molecular microbiology 104 (3) (2017)
Kenn Gerdes
Hypothesis: type I toxin-antitoxin genes enter the persistence field-a feedback mechanism explaining membrane homoeostasis. Philosophical transactions of the Royal Society of London. Series B, Biological sciences 371 (1707) (2017)
Kathryn J Turnbull, Kenn Gerdes
HicA toxin of Escherichia coli derepresses hicAB transcription to selectively produce HicB antitoxin. Molecular microbiology 104 (5) (2017)

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Publication Activity (10 Years)

Top Topics

Top Venues

Kenn Gerdes