Kentaro Semba

Publication Activity (10 Years)

Years Active: 2017-2023
Publications (10 Years): 7

Top Topics

Transcription Factor

Breast Cancer Cells

Top Venues

Genes to cells : devoted to molecular & cellular mechanisms

Science signaling

Molecular cancer research : MCR

Disease models & mechanisms

This page only lists publications with an associated author ORCID identifier.

Publications

Yuxuan Han, Shota Katayama, Mitsuru Futakuchi, Kazuya Nakamichi, Yutaro Wakabayashi, Mai Sakamoto, Jun Nakayama, Kentaro Semba
Targeting c-Jun is a potential therapy for luminal breast cancer bone metastasis. Molecular cancer research : MCR (2023)
Kazuya Nakamichi, Yusuke Yamamoto, Kentaro Semba, Jun Nakayama
Metastatic potentials classified with hypoxia-inducible factor 1 downstream genes in pan-cancer cell lines. Genes to cells : devoted to molecular & cellular mechanisms 29 (2) (2023)
Kazuya Nakamichi, Yusuke Yamamoto, Kentaro Semba, Jun Nakayama
Metastatic potentials classified with hypoxia-inducible factor 1 downstream genes in pan-cancer cell lines. Genes to cells : devoted to molecular & cellular mechanisms (2023)
Kazuya Nakamichi, Yusuke Yamamoto, Kentaro Semba, Jun Nakayama
Metastatic potentials classified with hypoxia-inducible factor 1 downstream genes in pan-cancer cell lines. Genes to cells : devoted to molecular & cellular mechanisms 29 (2) (2023)
Yuta Doi, Hiroaki Tagaya, Ayaka Noge, Kentaro Semba
Prediction of Resistance Mutations Against Upcoming Anaplastic Lymphoma Kinase Inhibitors. Targeted oncology 17 (6) (2022)
Jun Nakayama, Hiroko Matsunaga, Koji Arikawa, Takuya Yoda, Masahito Hosokawa, Haruko Takeyama, Yusuke Yamamoto, Kentaro Semba
Identification of two cancer stem cell-like populations in triple-negative breast cancer xenografts. Disease models & mechanisms 15 (6) (2022)
Yukari Okita, Minori Kimura, Rudy Xie, Chen Chen, Larina Tzu-Wei Shen, Yurika Kojima, Hiroyuki Suzuki, Masafumi Muratani, Masao Saitoh, Kentaro Semba, Carl-Henrik Heldin, Mitsuyasu Kato
The transcription factor MAFK induces EMT and malignant progression of triple-negative breast cancer cells through its target GPNMB. Science signaling 10 (474) (2017)