María Alcázar Fabra (María Alcazar-Fabra, María Alcázar-Fabra)

Publication Activity (10 Years)

Years Active: 2021-2024
Publications (10 Years): 10

Top Topics

Embryonic Stem Cells

Gene Expression

Top Venues

Nature communications

Science advances

Journal of inherited metabolic disease

BioFactors (Oxford, England)

This page only lists publications with an associated author ORCID identifier.

Publications

Mencía Espinosa-Martínez, María Alcázar Fabra, David Landeira
The molecular basis of cell memory in mammals: The epigenetic cycle. Science advances 10 (9) (2024)
Mencía Espinosa-Martínez, María Alcázar Fabra, David Landeira
The molecular basis of cell memory in mammals: The epigenetic cycle. Science advances 10 (9) (2024)
María Alcázar Fabra, Abraham J Paredes-Fuentes, Manuel Torralba Carnerero, Daniel José Moreno Fernández-Ayala, Antonio Arroyo Luque, Ana Sánchez-Cuesta, Carmine Staiano, Paula Sánchez-Pintos, Maria-Luz Couce, Miguel Tomás Vila, Ana Victoria Marco Hernández, Carmen Orellana, Francisco Martínez, Mónica Roselló, Alfonso Caro, Juan Silvestre Oltra Soler, Sandra Monfort, Alejandro Sánchez, Dolores Rausell, Isidro Vitoria, Mireia Del Toro, Ángeles García-Cazorla, Natalia A Julia-Palacios, Cristina Jou, Delia Yubero, Luis Carlos López, Juan Diego Hernández Camacho, Guillermo Lopez-Lluch, Manuel Ballesteros Simarro, Juan Carlos Rodríguez-Aguilera, Gloria Brea Calvo, María Victoria Cascajo Almenara, Rafael Artuch, Carlos Santos-Ocaña
New variants expand the neurological phenotype of COQ7 deficiency. Journal of inherited metabolic disease (2024)
Mencía Espinosa-Martínez, María Alcázar Fabra, David Landeira
The molecular basis of cell memory in mammals: The epigenetic cycle. Science advances 10 (9) (2024)
Helena G Asenjo, María Alcázar Fabra, Mencía Espinosa-Martínez, Lourdes Lopez-Onieva, Amador Gallardo, Emilia Dimitrova, Angelika Feldmann, Tomas Pachano, Jordi Martorell-Marugán, Pedro Carmona-Sáez, Antonio Sánchez Pozo, Álvaro Rada-Iglesias, Robert J Klose, David Landeira
Changes in PRC1 activity during interphase modulate lineage transition in pluripotent cells. Nature communications 14 (1) (2023)
Helena G Asenjo, María Alcázar Fabra, Mencía Espinosa-Martínez, Lourdes Lopez-Onieva, Amador Gallardo, Emilia Dimitrova, Angelika Feldmann, Tomas Pachano, Jordi Martorell-Marugán, Pedro Carmona-Sáez, Antonio Sánchez Pozo, Álvaro Rada-Iglesias, Robert J Klose, David Landeira
Changes in PRC1 activity during interphase modulate lineage transition in pluripotent cells. Nature communications 14 (1) (2023)
Helena G Asenjo, María Alcázar Fabra, Mencía Espinosa-Martínez, Lourdes Lopez-Onieva, Amador Gallardo, Emilia Dimitrova, Angelika Feldmann, Tomas Pachano, Jordi Martorell-Marugán, Pedro Carmona-Sáez, Antonio Sánchez Pozo, Álvaro Rada-Iglesias, Robert J Klose, David Landeira
Changes in PRC1 activity during interphase modulate lineage transition in pluripotent cells. Nature communications 14 (1) (2023)
Helena G Asenjo, María Alcázar Fabra, Mencía Espinosa-Martínez, Lourdes Lopez-Onieva, Amador Gallardo, Emilia Dimitrova, Angelika Feldmann, Tomas Pachano, Jordi Martorell-Marugán, Pedro Carmona-Sáez, Antonio Sánchez Pozo, Álvaro Rada-Iglesias, Robert J Klose, David Landeira
Changes in PRC1 activity during interphase modulate lineage transition in pluripotent cells. Nature communications 14 (1) (2023)
Helena G Asenjo, María Alcázar Fabra, Mencía Espinosa-Martínez, Lourdes Lopez-Onieva, Amador Gallardo, Emilia Dimitrova, Angelika Feldmann, Tomas Pachano, Jordi Martorell-Marugán, Pedro Carmona-Sáez, Antonio Sánchez Pozo, Álvaro Rada-Iglesias, Robert J Klose, David Landeira
Changes in PRC1 activity during interphase modulate lineage transition in pluripotent cells. Nature communications 14 (1) (2023)
Plácido Navas, María Victoria Cascajo Almenara, María Alcázar Fabra, Juan Diego Hernández Camacho, Ana Sánchez-Cuesta, Ana Belén Cortés Rodríguez, Manuel Ballesteros Simarro, Antonio Arroyo Luque, Juan Carlos Rodríguez-Aguilera, Daniel José Moreno Fernández-Ayala, Gloria Brea Calvo, Guillermo Lopez-Lluch, Carlos Santos-Ocaña
Secondary CoQ10 deficiency, bioenergetics unbalance in disease and aging. BioFactors (Oxford, England) 47 (4) (2021)