Oliver Wessely

Publication Activity (10 Years)

Years Active: 2018-2024
Publications (10 Years): 12

Top Topics

Diabetic Nephropathy

Top Venues

American journal of physiology. Renal physiology

bioRxiv : the preprint server for biology

This page only lists publications with an associated author ORCID identifier.

Publications

Uyen Tran, Andrew J Streets, Devon Smith, Eva Decker, Annemarie Kirschfink, Lahoucine Izem, Jessie M Hassey, Briana Rutland, Manoj K Valluru, Jan Hinrich Bräsen, Elisabeth Ott, Daniel Epting, Tobias Eisenberger, Albert Cm Ong, Carsten Bergmann, Oliver Wessely
BICC1 Interacts with PKD1 and PKD2 to Drive Cystogenesis in ADPKD. bioRxiv : the preprint server for biology (2024)
Sierra R McKinzie, Natalya Kaverina, Robert Allen Schweickart, Christopher P Chaney, Diana G Eng, Beatriz Maria Veloso Pereira, Bryan Kestenbaum, Jeffrey W Pippin, Oliver Wessely, Stuart J Shankland
Podocytes from hypertensive and obese mice acquire an inflammatory, senescent, and aged phenotype. American journal of physiology. Renal physiology 326 (4) (2024)
Sierra R McKinzie, Natalya Kaverina, Robert Allen Schweickart, Christopher P Chaney, Diana G Eng, Beatriz Maria Veloso Pereira, Bryan Kestenbaum, Jeffrey W Pippin, Oliver Wessely, Stuart J Shankland
Podocytes from hypertensive and obese mice acquire an inflammatory, senescent, and aged phenotype. American journal of physiology. Renal physiology 326 (4) (2024)
Sierra R McKinzie, Natalya Kaverina, Robert Allen Schweickart, Christopher P Chaney, Diana G Eng, Beatriz Maria Veloso Pereira, Bryan Kestenbaum, Jeffrey W Pippin, Oliver Wessely, Stuart J Shankland
Podocytes from hypertensive and obese mice acquire an inflammatory, senescent, and aged phenotype. American journal of physiology. Renal physiology 326 (4) (2024)
Sierra R McKinzie, Natalya Kaverina, Robert Allen Schweickart, Christopher P Chaney, Diana G Eng, Beatriz Maria Veloso Pereira, Bryan Kestenbaum, Jeffrey W Pippin, Oliver Wessely, Stuart J Shankland
Podocytes from hypertensive and obese mice acquire an inflammatory, senescent, and aged phenotype. American journal of physiology. Renal physiology 326 (4) (2024)
Sierra R McKinzie, Natalya Kaverina, Robert Allen Schweickart, Christopher P Chaney, Diana G Eng, Beatriz Maria Veloso Pereira, Bryan Kestenbaum, Jeffrey W Pippin, Oliver Wessely, Stuart J Shankland
Podocytes from hypertensive and obese mice acquire an inflammatory, senescent, and aged phenotype. American journal of physiology. Renal physiology 326 (4) (2024)
Sierra R McKinzie, Natalya Kaverina, Robert Allen Schweickart, Christopher P Chaney, Diana G Eng, Beatriz Maria Veloso Pereira, Bryan Kestenbaum, Jeffrey W Pippin, Oliver Wessely, Stuart J Shankland
Podocytes from hypertensive and obese mice acquire an inflammatory, senescent, and aged phenotype. American journal of physiology. Renal physiology 326 (4) (2024)
Sierra R McKinzie, Natalya Kaverina, Robert Allen Schweickart, Christopher P Chaney, Diana G Eng, Beatriz Maria Veloso Pereira, Bryan Kestenbaum, Jeffrey W Pippin, Oliver Wessely, Stuart J Shankland
Podocytes from hypertensive and obese mice acquire an inflammatory, senescent, and aged phenotype. American journal of physiology. Renal physiology 326 (4) (2024)
Sierra R McKinzie, Natalya Kaverina, Robert Allen Schweickart, Christopher P Chaney, Diana G Eng, Beatriz Maria Veloso Pereira, Bryan Kestenbaum, Jeffrey W Pippin, Oliver Wessely, Stuart J Shankland
Podocytes from hypertensive and obese mice acquire an inflammatory, senescent, and aged phenotype. American journal of physiology. Renal physiology 326 (4) (2024)
Stuart J Shankland, Andrew D Rule, J Nathan Kutz, Jeffrey W Pippin, Oliver Wessely
Podocyte Senescence and Aging. Kidney360 (2023)
Alyson Wolk, Dilara Hatipoglu, Alecia Cutler, Mariya Ali, Lestella Bell, Jian Hua Qi, Rupesh Singh, Julia Batoki, Laura Karle, Vera L Bonilha, Oliver Wessely, Heidi Stöhr, Vincent Hascall, Bela Anand-Apte
Role of FGF and Hyaluronan in Choroidal Neovascularization in Sorsby Fundus Dystrophy. Cells 9 (3) (2020)
Bo Zhang, Uyen Tran, Oliver Wessely
Polycystin 1 loss of function is directly linked to an imbalance in G-protein signaling in the kidney. Development (Cambridge, England) 145 (6) (2018)