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Histone deacetylase-mediated silencing of PSTPIP2 expression contributes to AAI-induced PANoptosis.

Chuanting XuQi WangChanglin DuJiahui DongZhenming ZhangNa CaiJun LiCheng HuangTaotao Ma
Published in: British journal of pharmacology (2023)
HDAC-mediated silencing of PSTPIP2 may contribute to aristolochic acid nephropathy. Hence, HDAC1 and -2 specific inhibitors or PSTPIP2 could be valuable therapeutic agents for preventing aristolochic acid nephropathy.
Keyphrases
  • histone deacetylase
  • poor prognosis
  • diabetic rats
  • oxidative stress
  • drug induced
  • endothelial cells