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High-intensity exercise training ameliorates aberrant expression of markers of mitochondrial turnover but not oxidative damage in skeletal muscle of men with essential hypertension.

Matteo FiorenzaThomas P GunnarssonThomas S EhlersJens Bangsbo
Published in: Acta physiologica (Oxford, England) (2018)
The present results indicate aberrant muscle mitochondrial turnover and augmented oxidative damage in hypertensive individuals. High-intensity exercise training can partly reverse hypertension-related impairments in muscle mitochondrial turnover, but not redox imbalance.
Keyphrases
  • high intensity
  • skeletal muscle
  • blood pressure
  • oxidative stress
  • resistance training
  • bone mineral density
  • insulin resistance
  • poor prognosis
  • binding protein
  • type diabetes
  • middle aged
  • long non coding rna