Repurposing Anakinra for Alzheimer's Disease: The In Vitro and In Vivo Effects of Anakinra on LPS- and AC-Induced Neuroinflammation.
Thaarvena RetinasamyAmber Lot Yee LeeHsien Siang LeeVanessa Lin Lin LeeMohd Farooq ShaikhKeng Yoon YeongPublished in: ACS chemical neuroscience (2024)
Alzheimer's disease is a significant global health issue, and studies suggest that neuroinflammation plays a vital role in the advancement of this disease. In this study, anakinra has been shown to display a time- and concentration-dependent antineuroinflammatory effect. In the in vitro studies, it diminished the gene expressions of tumor necrosis factor-alpha (TNF-α) and nitric oxide (NO) synthase 2 stimulated by lipopolysaccharide (LPS). Anakinra also reduced the LPS-induced production of NO and reactive oxygen species. Thus, the hypertrophic state of LPS-activated BV2 microglial cells was reversed by anakinra. Furthermore, acrylamide (ACR)-induced activation of nuclear transcription factor-κB, TNF-α, and interleukin-1β was downregulated, while cAMP response element binding protein and brain-derived neurotrophic factor expression levels were markedly enhanced in ACR-treated zebrafish larvae. It was also observed that anakinra improved the uncoordinated swimming behaviors in ACR-exposed zebrafish larvae. Overall, anakinra demonstrated potential antineuroinflammatory and antioxidative effects.
Keyphrases
- lps induced
- inflammatory response
- lipopolysaccharide induced
- binding protein
- rheumatoid arthritis
- toll like receptor
- nitric oxide
- global health
- transcription factor
- anti inflammatory
- reactive oxygen species
- high glucose
- traumatic brain injury
- public health
- diabetic rats
- induced apoptosis
- hydrogen peroxide
- cell death
- cognitive impairment
- stress induced
- signaling pathway
- cerebral ischemia
- cell cycle arrest
- case control
- spinal cord injury
- climate change
- neuropathic pain
- dna methylation
- nitric oxide synthase