Login / Signup

Modulation of the microhomology-mediated end joining pathway suppresses large deletions and enhances homology-directed repair following CRISPR-Cas9-induced DNA breaks.

Baolei YuanChongwei BiYeteng TianJincheng WangYiqing JinKhaled AlsayeghMuhammad TehseenGang YiXuan ZhouYanjiao ShaoFernanda Vargas RomeroWolfgang FischleJuan Carlos Izpisua BelmonteSamir HamdanYanyi HuangMo Li
Published in: BMC biology (2024)
Our findings reveal the contrasting roles of RPA and POLQ in Cas9-induced LD and HDR, suggesting new strategies for safer and more precise genome editing.
Keyphrases
  • crispr cas
  • genome editing
  • high glucose
  • diabetic rats
  • drug induced
  • signaling pathway
  • single molecule
  • single cell
  • dna repair
  • circulating tumor
  • gene expression
  • dna damage
  • circulating tumor cells