TNS may prevent brain damage and relieve neurological deficits after a TBI by inhibiting inflammation, possibly via the TLR4/NF-κB/NLRP3 signaling pathway mediated by OX-A/OX1R.
Keyphrases
- signaling pathway
- traumatic brain injury
- pi k akt
- toll like receptor
- oxidative stress
- induced apoptosis
- inflammatory response
- epithelial mesenchymal transition
- immune response
- nuclear factor
- severe traumatic brain injury
- low density lipoprotein
- spinal cord injury
- lps induced
- white matter
- nlrp inflammasome
- neuropathic pain
- resting state
- cell proliferation
- mild traumatic brain injury
- subarachnoid hemorrhage