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Interleukin-1 alpha derived from ultraviolet B-exposed keratinocytes is associated with a decrease of endocytic collagen receptor Endo180.

Hiroyasu IwahashiYoshihito KawashimaHitoshi Masaki
Published in: Photodermatology, photoimmunology & photomedicine (2019)
These results indicate that a substance secreted from UVB-exposed keratinocytes regulates Endo180 expression and that IL-1α may play an important role in the maintenance of Endo180.
Keyphrases
  • wound healing
  • poor prognosis
  • binding protein