Candidate genes for polycystic ovary syndrome are regulated by TGFβ in the bovine foetal ovary.
Rafiatu AzumahMenghe LiuKatja HummitzschNicole A BastianMonica Dwi HartantiHelen F Irving-RodgersRichard A AndersonRaymond J RodgersPublished in: Human reproduction (Oxford, England) (2022)
Funding was received from Adelaide University China Fee Scholarship (M.L.), Australian Research Training Program (R.A.) and the Faculty of Health and Medical Science Divisional Scholarship (R.A.), Adelaide Graduate Research Scholarships (R.A. and N.A.B.), Australia Awards Scholarship (M.D.H.), Robinson Research Institute Career Development Fellowship (K.H.) and Building On Ideas Grant (K.H.), National Health and Medical Research Council of Australia Centre for Research Excellence in the Evaluation, Management and Health Care Needs of Polycystic Ovary Syndrome (N.A.B., M.D.H. and R.J.R.; GTN1078444) and the Centre for Research Excellence on Women's Health in Reproductive life (R.A., R.J.R. and K.H.; GTN1171592) and the UK Medical Research Council (R.A.A.; grant no. G1100357). The funders did not play any role in the study design, data collection and analysis, decision to publish or preparation of the manuscript. The authors of this manuscript have nothing to declare and no conflict of interest that could be perceived as prejudicing the impartiality of the research reported.
Keyphrases
- polycystic ovary syndrome
- healthcare
- insulin resistance
- public health
- mental health
- health information
- emergency medicine
- medical students
- adipose tissue
- transforming growth factor
- metabolic syndrome
- electronic health record
- depressive symptoms
- medical education
- physical activity
- type diabetes
- cross sectional
- big data
- skeletal muscle
- epithelial mesenchymal transition
- high resolution
- pregnant women
- artificial intelligence
- human health