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Inhibition of GPR91 Reduces Inflammatory Mediators Involved in Active Labor in Myometrium.

Ratana LimMartha Lappas
Published in: Mediators of inflammation (2020)
Our findings demonstrate that GPR91 is involved in the genesis of proinflammatory and prolabor mediators induced by IL1B or TNF and collectively suggest that GPR91 may contribute to augmentation of the labor processes.
Keyphrases
  • fatty acid
  • rheumatoid arthritis
  • oxidative stress
  • soft tissue