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Activation of metabolic and stress responses during subtoxic expression of the type I toxin hok in Erwinia amylovora.

Jingyu PengLindsay R TriplettGeorge W Sundin
Published in: BMC genomics (2021)
We characterized the molecular mechanism of toxicity by high-level of hok induction and demonstrated that low-level expression of hok primes the stress responses of E. amylovora against further membrane and antibiotic stressors.
Keyphrases
  • poor prognosis
  • escherichia coli