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Preservation of β-cell function in pancreatic insufficient cystic fibrosis with highly effective CFTR modulator therapy.

Anneliese Js FlattSaba SheikhAmy J PeleckisPaola AlvaradoDenis HadjiliadisDarko StefanovskiRobert J GallopRonald C RubensteinAndrea KellyMichael R Rickels
Published in: The Journal of clinical endocrinology and metabolism (2023)
ETI preserves β-cell function in CF through effects on glucose-dependent insulin secretion, proinsulin processing and meal-related insulin secretion. Further work should determine whether early intervention with ETI can prevent deterioration of glucose tolerance in PI-CF.
Keyphrases
  • cystic fibrosis
  • pseudomonas aeruginosa
  • lung function
  • randomized controlled trial
  • blood glucose
  • blood pressure
  • metabolic syndrome
  • type diabetes
  • mesenchymal stem cells
  • adipose tissue
  • insulin resistance