Tetraethylammonium, glibenclamide, and 4-aminopyridine modulate post-occlusive reactive hyperemia in non-glabrous human skin with no roles of NOS and COX.
Naoto FujiiGregory W McGarrMasashi IchinoseTakeshi NishiyasuGlen P KennyPublished in: Microcirculation (New York, N.Y. : 1994) (2019)
We showed that tetraethylammonium, glibenclamide, and 4-aminopyridine modulate PORH with no roles of NOS and COX in human non-glabrous forearm skin in vivo. Thus, cutaneous PORH changes could reflect altered K+ channel function.