ERS contributes to the pathophysiology of GDM in pregnancy via the CHOP-PPARα-NF-κB-signalling pathway by inducing aberrant activation of inflammation and insulin resistance.
Keyphrases
- endoplasmic reticulum stress
- insulin resistance
- induced apoptosis
- oxidative stress
- signaling pathway
- diffuse large b cell lymphoma
- lps induced
- adipose tissue
- metabolic syndrome
- high fat diet
- pregnancy outcomes
- pi k akt
- skeletal muscle
- nuclear factor
- polycystic ovary syndrome
- type diabetes
- pregnant women
- high fat diet induced
- preterm birth
- fatty acid
- glycemic control
- toll like receptor
- immune response