Fatty acid-binding protein 3 controls contact hypersensitivity through regulating skin dermal Vγ4+ γ/δ T cell in a murine model.
Shuhei KobayashiHai The PhungYoshiteru KagawaHirofumi MiyazakiYu TakahashiAtsuko AsaoTakashi MaruyamaAkihiko YoshimuraNaoto IshiiYuji OwadaPublished in: Allergy (2020)
These findings suggest that FABP3 functions as a negative regulator of skin inflammation through limiting pathogenic Vγ4+ γ/δ T-cell generation in the thymus.