Rational Design and Identification of Harmine-Inspired, N-Heterocyclic DYRK1A Inhibitors Employing a Functional Genomic In Vivo Drosophila Model System.
Francisco J HuizarHarrison M HillEmily P BacherKaitlyn E EckertEva M GulottyKevin X RodriguezZachary D TuckerMonimoy BanerjeeHaining LiuOlaf G WiestJeremiah J ZartmanBrandon L AshfeldPublished in: ChemMedChem (2022)
Deregulation of dual-specificity tyrosine phosphorylation-regulated kinase 1A (DYRK1A) plays a significant role in developmental brain defects, early-onset neurodegeneration, neuronal cell loss, dementia, and several types of cancer. Herein, we report the discovery of three new classes of N-heterocyclic DYRK1A inhibitors based on the potent, yet toxic kinase inhibitors, harmine and harmol. An initial in vitro evaluation of the small molecule library assembled revealed that the core heterocyclic motifs benzofuranones, oxindoles, and pyrrolones, showed statistically significant DYRK1A inhibition. Further, the utilization of a low cost, high-throughput functional genomic in vivo model system to identify small molecule inhibitors that normalize DYRK1A overexpression phenotypes is described. This in vivo assay substantiated the in vitro results, and the resulting correspondence validates generated classes as architectural motifs that serve as potential DYRK1A inhibitors. Further expansion and analysis of these core compound structures will allow discovery of safe, more effective chemical inhibitors of DYRK1A to ameliorate phenotypes caused by DYRK1A overexpression.
Keyphrases
- protein protein
- small molecule
- early onset
- high throughput
- low cost
- single cell
- cell proliferation
- transcription factor
- late onset
- multiple sclerosis
- cognitive impairment
- mass spectrometry
- squamous cell carcinoma
- copy number
- cell therapy
- blood brain barrier
- subarachnoid hemorrhage
- cerebral ischemia
- bone marrow
- tyrosine kinase
- resting state