High expression of SLC26A6 in the kidney may contribute to renal calcification via an SLC26A6-dependent mechanism.

Hongyang JiangGaurab PokhrelYinwei ChenTao WangChunping YinJihong LiuShaogang WangZhuo Liu

Published in: PeerJ (2018)

The results showed that high expression levels of renal SLC26A6 may account for kidney stone formation. Downregulating the expression of SLC26A6 in the kidney may be a potential therapeutic target to prevent or treat urolithiasis.

Keyphrases

poor prognosis
binding protein
long non coding rna
chronic kidney disease
risk assessment