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Cigarette smoke and electronic cigarettes differentially activate bronchial epithelial cells.

Christian HerrKonstantinos TsitourasJulia NiederstraßerChristina BackesChristoph BeisswengerLi DongLoïc GuillotAndreas KellerRobert Bals
Published in: Respiratory research (2020)
The transcriptome patterns of host defense and inflammatory genes are significantly distinct between ECIG-exposed and TCIG-treated cells. The overall effects of ECIGs on epithelial cells are less in comparison to TCIG, and ECIG-vapor does not affect host defense. Nevertheless, although acute exposure to ECIG-vapor induces inflammation, and the expression of S100 proteins, long term in vivo data is needed to evaluate the chronic effects of ECIG use.
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