Diabetes-Mediated Toxicity Resulted in the Expression of CD80 and CD86 on Neutrophils after Delayed Wound Healing in Male Rats.

Hossam EbaidBahaa Abdel-SalamIftekhar HassanJameel Al-TamimiAlli MetwalliAhmed RadyIbrahim M Alhazza

Published in: BioMed research international (2020)

Prolonged wound stress induced toxicity in DM rats and significantly increased the expression of CD80 and CD86 on PMNs. CMP peptide ameliorated the levels of toxicity markers, CD80 and CD86, and chemoattractant molecules in diabetic rats.

Keyphrases

stress induced
oxidative stress
poor prognosis
nk cells
diabetic rats
type diabetes
cardiovascular disease
metabolic syndrome
skeletal muscle
adipose tissue
binding protein
insulin resistance
glycemic control