Endoplasmic reticulum stress-induced release and binding of calreticulin from human ovarian cancer cells.

Trefa M AbdullahJacqueline L WhatmoreEdwin BremerRimantas SlibinskasMarek MichalakEdwin Bremer

Published in: Cancer immunology, immunotherapy : CII (2021)

Collectively, these cellular responses suggest that ER stress is partially responsible for the changes in CRT cellular distribution. ER-stress regulates in part the release and binding of CRT to human OC cells where it may play a role in ICD.

Keyphrases

endoplasmic reticulum stress
induced apoptosis
endothelial cells
high glucose
induced pluripotent stem cells
pluripotent stem cells
signaling pathway
oxidative stress
heart failure
cardiac resynchronization therapy
dna binding
transcription factor
atrial fibrillation
cell death