Glycolysis-Mediated Activation of v-ATPase by Nicotinamide Mononucleotide Ameliorates Lipid-Induced Cardiomyopathy by Repressing the CD36-TLR4 Axis.
Shujin WangYinying HanRuimin LiuMengqian HouDietbert NeumannJun ZhangFang WangYumeng LiXueya ZhaoFrancesco SchianchiChao DaiLi-Zhong LiuMiranda NabbenJan F C GlatzXin WuXifeng LuXi LiJoost J F P LuikenPublished in: Circulation research (2024)
NMN preserves heart function during lipid overload by preventing v-ATPase disassembly.