Helicobacter pylori sabA gene is associated with iron deficiency anemia in childhood and adolescence.
Seiichi KatoTakako OsakiShigeru KamiyaXue-Song ZhangMartin J BlaserPublished in: PloS one (2017)
It is likely that H. pylori carrying high expression of sabA causes IDA, especially in children and adolescents who have increased daily iron demand. In addition, it is possible that several host-interactive genes, including vacA, may play a synergistic role for sabA in IDA development.
Keyphrases
- iron deficiency
- helicobacter pylori
- genome wide
- genome wide identification
- helicobacter pylori infection
- poor prognosis
- depressive symptoms
- copy number
- physical activity
- genome wide analysis
- dna methylation
- cancer therapy
- transcription factor
- gene expression
- binding protein
- long non coding rna
- chronic kidney disease
- childhood cancer