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Molecular Mechanisms Linking Empagliflozin to Renal Protection in the LLC-PK1 Model of Diabetic Nephropathy.

Vjera NincevicMilorad ZjalicTomislav KizivatTea Omanovic KolaricMartina SmolicEdi RođakMarina ČovićLucija Kuna RoguljicRobert SmolicAleksandar VčevInes Bilić Ćurčić
Published in: Biomedicines (2022)
Renoprotective effects of empagliflozin, in this LLC-PK1 cell model of DN, are mediated via activation of the Akt/GSK-3 signalling pathway, thus reducing oxidative stress-induced damage, as well as enhanced SMAD7 expression leading to downregulation of TGF-β1, one of the key mediators of inflammation and fibrosis.
Keyphrases
  • diabetic nephropathy
  • signaling pathway
  • oxidative stress
  • transforming growth factor
  • cell proliferation
  • poor prognosis
  • epithelial mesenchymal transition
  • single cell
  • pi k akt
  • stem cells
  • cell therapy
  • binding protein