Evaluating mRNA expression of tax, B chain of PDGF and PDGF-β receptors as well as HTLV-I proviral load in ATL patients and healthy carriers.
Hadi AtabatiSeyed-Alireza EsmaeiliAbolghasem AllahyariAbbas ShirdelHossein RahimiSeyed Abdolrahim RezaeeAmir Abbas Momtazi-BorojeniHoushang RafatpanahPublished in: Journal of medical virology (2020)
Adult T-cell leukemia (ATL) is a life-threatening malignant neoplasm of CD4+ T cells resulted from human T-cell leukemia virus type I (HTLV-I). Tax1 protein of HTLV-I can induce malignant proliferation of T-cells by modulating the expression of growth factors such as platelet-derived growth factor (PDGF). Here, we aimed to investigate the proviral load (PVL) of HTLV-I in ATL and also to evaluate the mRNA expression of B chain of PDGF and PDGF-β receptors in ATL patients and HTLV-I-infected healthy carriers. To this end, peripheral blood mononuclear cells (PBMCs) were isolated by using Ficoll-Histophaque density centrifugation. The mean of HTLV-I PVL in ATL patients (42,759 ± 15,737 copies/104 cells [95% CI, 9557-75962]) was significantly (p = .01) higher than that in healthy carriers (650 ± 107 copies/104 cells [95% CI, 422-879], respectively. The HTLV-I PVL in ATL patients exhibited a significant correlation with PBMC count (R = .495, p = .001). The mRNA expression of Tax, B chain of PDGF, and PDGF-β receptor genes was significantly higher in healthy carriers than in patients with ATL. In conclusion, the expression of the canonical PDGFβ and its receptor, and their correlation with Tax expression cannot be a suitable indicator and/or prognostic factor for progression of ATL in HTLV-I carriers.
Keyphrases
- prognostic factors
- end stage renal disease
- ejection fraction
- newly diagnosed
- chronic kidney disease
- growth factor
- vascular smooth muscle cells
- poor prognosis
- induced apoptosis
- acute myeloid leukemia
- signaling pathway
- dna methylation
- bone marrow
- oxidative stress
- genome wide
- long non coding rna
- cell proliferation
- peripheral blood
- patient reported
- gene expression
- angiotensin ii
- small molecule
- amino acid