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ROS Induction Targets Persister Cancer Cells with Low Metabolic Activity in NRAS-Mutated Melanoma.

Ossia M EichhoffCorinne I StoffelJan KäslerLuzia BrikerPatrick TurkoGergely KarsaiNina ZilaVerena PaulitschkePhil Fang ChengAlexander LeitnerAndrea BileckNicola ZamboniAnja IrmischZsolt BalazsAizhan TastanovaSusana PascoalPål JohannsenRebekka WegmannJulien MenaAlaa OthmanVasanthi S ViswanathanJudith WenzinaAndrea AloiaAnnalisa SaltariAndreas Dzungnull nullMichael KrauthammerStuart L SchreiberThorsten HornemannMartin DistelBerend SnijderReinhard DummerMitchell Paul Levesque
Published in: Cancer research (2023)
Metabolic reprogramming in drug-resistant NRAS-mutated melanoma cells confers sensitivity to ROS induction, which suppresses tumor growth and metastasis in combination with MAPK pathway inhibitors.
Keyphrases
  • drug resistant
  • wild type
  • multidrug resistant
  • acinetobacter baumannii
  • signaling pathway
  • cell death
  • dna damage
  • reactive oxygen species
  • oxidative stress
  • pi k akt
  • pseudomonas aeruginosa