SYT7 acts as an oncogene and a potential therapeutic target and was regulated by ΔNp63α in HNSCC.

You FuGuocai TianZhiyuan ZhangXiao Yang

Published in: Cancer cell international (2021)

Taken together, we found that ΔNp63α could inhibit the occurrence and progression of HNSCC throughout downregulating the expression of SYT7. Therefore, SYT7/ΔNp63α axis could be a potential therapeutic target for clinical treatment of HNSCC.

Keyphrases

poor prognosis
risk assessment
human health
binding protein
long non coding rna
combination therapy
climate change