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Genome-wide analysis of multi- and extensively drug-resistant Mycobacterium tuberculosis.

Francesc CollJody PhelanGrant A Hill-CawthorneMridul B NairKim MallardShahjahan AliAbdallah M AbdallahSaad AlghamdiMona AlsomaliAbdallah O AhmedStephanie PortelliYaa OppongAdriana AlvesTheolis Barbosa BessaSusana CampinoMaxine CawsAnirvan ChatterjeeAmelia C CrampinKeertan DhedaNicholas FurnhamJudith R GlynnLouis GrandjeanDang Minh HaRumina HasanZahra HasanMartin L HibberdMoses JolobaEdward C Jones-LópezTomoshige MatsumotoAnabela MirandaDavid A J MooreNora MocilloStefan PanaiotovJulian ParkhillCarlos PenhaJoão PerdigãoIsabel PortugalZineb RchiadJaime RobledoPatricia SheenNashwa Talaat SheshaFrik A SirgelChristophe SolaErivelton Oliveira SousaElizabeth M StreicherPaul Van HeldenMiguel ViveirosRobert M WarrenRuth McNerneyArnab PainTaane Gregory Clark
Published in: Nature genetics (2018)
To characterize the genetic determinants of resistance to antituberculosis drugs, we performed a genome-wide association study (GWAS) of 6,465 Mycobacterium tuberculosis clinical isolates from more than 30 countries. A GWAS approach within a mixed-regression framework was followed by a phylogenetics-based test for independent mutations. In addition to mutations in established and recently described resistance-associated genes, novel mutations were discovered for resistance to cycloserine, ethionamide and para-aminosalicylic acid. The capacity to detect mutations associated with resistance to ethionamide, pyrazinamide, capreomycin, cycloserine and para-aminosalicylic acid was enhanced by inclusion of insertions and deletions. Odds ratios for mutations within candidate genes were found to reflect levels of resistance. New epistatic relationships between candidate drug-resistance-associated genes were identified. Findings also suggest the involvement of efflux pumps (drrA and Rv2688c) in the emergence of resistance. This study will inform the design of new diagnostic tests and expedite the investigation of resistance and compensatory epistatic mechanisms.
Keyphrases
  • mycobacterium tuberculosis
  • drug resistant
  • genome wide
  • genome wide association study
  • dna methylation
  • copy number
  • drug induced