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Chronic glucokinase activator treatment activates liver Carbohydrate response element binding protein and improves hepatocyte ATP homeostasis during substrate challenge.

Brian E FordShruti S ChachraAhmed AlshawiAlfie BrennanSuzannah J HarnorCeline CanoDavid J BakerDavid M SmithRebecca J FaircloughLoranne Agius
Published in: Diabetes, obesity & metabolism (2020)
Chronic GKA treatment of C57BL/6 mice for 8 weeks activates liver ChREBP and improves the resilience of hepatocytes to compromised ATP homeostasis during high-substrate challenge. These changes are associated with raised mRNA levels of ChREBP-β and both catalytic subunits of AMP-activated protein kinase.
Keyphrases
  • protein kinase
  • binding protein
  • type diabetes
  • climate change
  • immune response
  • combination therapy
  • social support
  • adipose tissue
  • toll like receptor
  • inflammatory response
  • gestational age