Crystal structures of BMPRII extracellular domain in binary and ternary receptor complexes with BMP10.
Jingxu GuoBin LiuMidory ThorikayMinmin YuXiaoyan LiZhen TongRichard M SalmonRandy J ReadPeter Ten DijkeNicholas W MorrellWei LiPublished in: Nature communications (2022)
Heterozygous mutations in BMPR2 (bone morphogenetic protein (BMP) receptor type II) cause pulmonary arterial hypertension. BMPRII is a receptor for over 15 BMP ligands, but why BMPR2 mutations cause lung-specific pathology is unknown. To elucidate the molecular basis of BMP:BMPRII interactions, we report crystal structures of binary and ternary BMPRII receptor complexes with BMP10, which contain an ensemble of seven different BMP10:BMPRII 1:1 complexes. BMPRII binds BMP10 at the knuckle epitope, with the A-loop and β4 strand making BMPRII-specific interactions. The BMPRII binding surface on BMP10 is dynamic, and the affinity is weaker in the ternary complex than in the binary complex. Hydrophobic core and A-loop interactions are important in BMPRII-mediated signalling. Our data reveal how BMPRII is a low affinity receptor, implying that forming a signalling complex requires high concentrations of BMPRII, hence mutations will impact on tissues with highest BMPR2 expression such as the lung vasculature.
Keyphrases
- pulmonary arterial hypertension
- mesenchymal stem cells
- bone regeneration
- pulmonary hypertension
- pulmonary artery
- binding protein
- ionic liquid
- bone marrow
- transcription factor
- early onset
- poor prognosis
- coronary artery
- dna methylation
- machine learning
- deep learning
- artificial intelligence
- convolutional neural network
- data analysis