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Multi-Omic Architecture of Obstructive Hypertrophic Cardiomyopathy.

Ramin GarmanyJ Martijn BosDavid J TesterJohn R GiudicessiCristobal G Dos RemediosSurendra DasariNagaswaroop K NagarajAsha A NairKenneth L JohnsonZachary C RyanJoseph J MaleszewskiSteven R OmmenJoseph A DearaniMichael J Ackerman
Published in: Circulation. Genomic and precision medicine (2023)
At time of surgical myectomy, the ventricular proteome, independent of genotype, reveals widespread upregulation and activation of hypertrophy pathways, mainly involving the RAS-MAPK signaling cascade. In addition, there is a counterregulatory transcriptional downregulation of the same pathways. RAS-MAPK activation may serve a crucial role in hypertrophy observed in HCM.
Keyphrases
  • hypertrophic cardiomyopathy
  • left ventricular
  • signaling pathway
  • pi k akt
  • cell proliferation
  • oxidative stress
  • wild type
  • heart failure
  • gene expression
  • transcription factor
  • poor prognosis
  • heat shock