Hydrogen Sulphide Treatment Prevents Renal Ischemia-Reperfusion Injury by Inhibiting the Expression of ICAM-1 and NF-kB Concentration in Normotensive and Hypertensive Rats.
Syed F HashmiHassaan Anwer RathoreMunavvar A SattarEdward J JohnsChee Yuen GanTan Yong ChiaAshfaq AhmadPublished in: Biomolecules (2021)
Our main objective was to investigate the effect of chronic administration of hydrogen sulphide donor (sodium hydrosulphide) on the expression of intercellular adhesion molecule-1 (ICAM-1) and concentration of nuclear factor-kappa B (NF-kB) in a renal ischemia-reperfusion injury (IRI) model of WKY and L-nitro-arginine-methyl-ester (L-NAME)-induced hypertensive rats. Sodium hydrosulphide (NaHS) was administered intraperitoneally (i.p.) for 35 days while cystathionine gamma lyase (CSE) inhibitor dL-propargylglycine (PAG) was administered at a single dose of 50 mg/kg. Animals were anesthetised using sodium pentobarbitone (60 mg/kg) and then prepared to induce renal ischemia by clamping the left renal artery for 30 min followed by 3 h of reperfusion. Pre-treatment with NaHS improved the renal functional parameters in both WKY and L-NAME-induced hypertensive rats along with reduction of blood pressure in hypertensive groups. Oxidative stress markers like malondialdehyde (MDA), total superoxide dismutase (T-SOD) and glutathione (GSH) were also improved by NaHS treatment following renal IRI. Levels of ICAM-1 and NF-kB concentration were reduced by chronic treatment with NaHS and increased by PAG administration after renal IRI in plasma and kidney. Treatment with NaHS improved tubular morphology and glomerulus hypertrophy. Pre-treatment with NaHS reduced the degree of renal IRI by potentiating its antioxidant and anti-inflammatory mechanism, as evidenced by decreased NF-kB concentration and downregulation of ICAM-1 expression.
Keyphrases
- nuclear factor
- oxidative stress
- blood pressure
- signaling pathway
- ischemia reperfusion injury
- poor prognosis
- anti inflammatory
- diabetic rats
- lps induced
- escherichia coli
- toll like receptor
- heart failure
- dna damage
- left ventricular
- immune response
- nitric oxide
- heart rate
- binding protein
- hydrogen peroxide
- insulin resistance
- heat shock
- blood glucose
- endoplasmic reticulum stress
- cell migration
- acute ischemic stroke
- skeletal muscle
- amino acid
- smoking cessation