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Modulation of hepcidin expression by normal control and beta0-thalassemia/Hb E erythroblasts.

Janejira JaratsittisinWannapa SornjaiSaovaros SvastiSuthat FucharoenSittiruk RoytrakulDuncan R Smith
Published in: Hematology (Amsterdam, Netherlands) (2017)
The in vitro cell system developed here allows further investigation into the processes mediating erythroid cell suppression of liver hepcidin expression in both normal and pathological states.
Keyphrases
  • poor prognosis
  • single cell
  • cell therapy
  • binding protein
  • stem cells
  • bone marrow
  • sickle cell disease