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Heat shock protein 27 deficiency promotes ferrous ion absorption and enhances acyl-Coenzyme A synthetase long-chain family member 4 stability to promote glioblastoma cell ferroptosis.

Kai ZhangYue WuGuangliang ChenHao WangYongsheng LiuYouxin Zhou
Published in: Cancer cell international (2023)
HSP27 deficiency promotes GBM ferroptosis. Targeting HSP27 may serve as a new direction for GBM treatment.
Keyphrases
  • heat shock protein
  • cell death
  • heat shock
  • replacement therapy
  • single cell
  • cancer therapy
  • fatty acid
  • combination therapy
  • bone marrow
  • smoking cessation
  • mesenchymal stem cells