Phosphorylation in Novel Mitochondrial Creatine Kinase Tyrosine Residues Render Cardioprotection against Hypoxia/Reoxygenation Injury.

Nammi Park Jubert Marquez Maria Victoria Faith Garcia Ippei Shimizu Sung Ryul Lee Hyoung Kyu Kim Jin Han

Published in: Journal of lipid and atherosclerosis (2021)

These results suggest that regulation of quantitative expression and phosphorylation site Y368 of CKMT2 offers a unique mechanism in future ICM therapeutics.

Keyphrases

protein kinase
poor prognosis
oxidative stress
small molecule
current status
induced apoptosis
high resolution
binding protein
signaling pathway