Excess Iodine Consumption Induces Oxidative Stress and Pancreatic Damage Independently of Chemical Form in Male Wistar Rats: Participation of PPAR-γ and C/EBP-β.
Cristian Arbez-EvangelistaOmar Arroyo-XochihuaIlse Haide Ortega-IbarraEdú Ortega-IbarraDe León-Ramírez Yeimy MarEstela Cuevas RomeroOmar Arroyo-HelgueraPublished in: Biology (2024)
The pancreatic damage, hypertriglyceridemia, and oxidative stress were independent of the chemical form of iodine consumed. These effects depended on PPAR-γ, C/EBP-β, GLUT-4, and IR.