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Endothelial DLL4 Is an Adipose Depot-Specific Fasting Sensor Regulating Fatty Acid Fluxes.

Alex AupetitPauline DecaunesChloé BellesElodie RiantJean GalitzkyCandice ChapoulyMargaux LaisnéRémy Flores-FloresBenoit ChaputKatell ViéJean-François ArnalAnne BouloumiéAnaïs Briot
Published in: Arteriosclerosis, thrombosis, and vascular biology (2023)
DLL4 is a major actor in the adaptive mechanisms of AT-EC to regulate lipid fluxes. It likely contributes to fat depot-dependent metabolism in response to energy transition states. AT-EC alteration with obesity may favor metabolic inflexibility and the development of cardiometabolic disorders.
Keyphrases
  • fatty acid
  • insulin resistance
  • adipose tissue
  • metabolic syndrome
  • high fat diet induced
  • type diabetes
  • skeletal muscle
  • weight loss
  • endothelial cells
  • weight gain
  • blood glucose