Effects of pepsin A on heat shock protein 70 response in laryngopharyngeal reflux patients with chronic rhinosinusitis.

Jing WangZhao YuJianjun RenYang XuYuke ZhangLei LeiYongbo ZhengLigao HuangZhaoping He

Published in: Acta oto-laryngologica (2017)

These results suggest that LPR may play a role in the development of CRS through pepsin A reflux, and increased HSP70 expression may be associated with the pathogenic mechanism of mucosal injury in CRS.

Keyphrases

heat shock protein
chronic rhinosinusitis
poor prognosis
heat shock
ulcerative colitis
binding protein
long non coding rna
oxidative stress