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BMP6 knockdown enhances cardiac fibrosis in a mouse myocardial infarction model by upregulating AP-1/CEMIP expression.

Guiping LuZhuowang GeXinyuan ChenYue MaAncai YuanYuquan XieJun Pu
Published in: Clinical and translational medicine (2023)
Therefore, BMP6 may be a novel molecular target for improving myocardial fibrosis and cardiac function after myocardial infarction.
Keyphrases
  • left ventricular
  • mesenchymal stem cells
  • poor prognosis
  • heart failure
  • bone regeneration
  • liver fibrosis
  • binding protein
  • bone marrow
  • single molecule