The present study provides further evidence of the relevance of the arachidonic acid cascade as an essential part of neuroinflammation in the pathology of ASD.
Keyphrases
- autism spectrum disorder
- mouse model
- attention deficit hyperactivity disorder
- intellectual disability
- binding protein
- poor prognosis
- white matter
- traumatic brain injury
- lipopolysaccharide induced
- resting state
- inflammatory response
- lps induced
- multiple sclerosis
- functional connectivity
- cognitive impairment
- subarachnoid hemorrhage
- brain injury
- working memory