Flavivirus prM interacts with MDA5 and MAVS to inhibit RLR antiviral signaling.

Liyan SuiYinghua ZhaoWenfang WangHongmiao ChiTian TianPing WuJinlong ZhangYicheng ZhaoZheng-Kai WeiZhijun HouGuoqiang ZhouGuoqing WangZedong WangQuan Liu

Published in: Cell & bioscience (2023)

Our findings reveal the immune evasion mechanisms of flavivirus prM, which may contribute to understanding flavivirus pathogenicity, therapeutic intervention and vaccine development.

Keyphrases

randomized controlled trial
breast cancer cells
single cell
biofilm formation
binding protein
gene expression
dna methylation
cell death
cell cycle arrest
cystic fibrosis