Long non-coding RNA FAL1 functions as a ceRNA to antagonize the effect of miR-637 on the down-regulation of AKT1 in Hirschsprung's disease.
Yang LiLingling ZhouChanggui LuQiyang ShenYang SuZhengke ZhiFeng WuHua ZhangZechao WenGuanglin ChenHongxing LiYankai XiaWeibing TangPublished in: Cell proliferation (2018)
These results illuminated that FAL1 may work as a ceRNA to modulate AKT1 expression via competitively binding to miR-637 in HSCR, suggesting that it may be clinically valuable as a biomarker of HSCR.