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Selective inhibition of PKR by C16 accelerates diabetic wound healing by inhibiting NALP3 expression in mice.

Kalyani KarnamKavitha SedmakiPravesh SharmaVenkata Vamsi Krishna VenugantiOnkar Prakash Kulkarni
Published in: Inflammation research : official journal of the European Histamine Research Society ... [et al.] (2022)
PKR inhibition using C16 accelerates the wound healing process in diabetic mice by decreasing NALP3-mediated IL-1β maturation.
Keyphrases
  • wound healing
  • poor prognosis
  • signaling pathway
  • type diabetes
  • high fat diet induced
  • metabolic syndrome