Differential capability of metabolic substrates to promote hepatocellular lipid accumulation.

Ngoc Anh HoangFriederike RichterMartin SchubertStefan LorkowskiLars-Oliver KlotzHolger Steinbrenner

Published in: European journal of nutrition (2018)

Several treatment regimens of excessive fat and sugar supply promoted lipid accumulation in HepG2 cells, albeit with differences in the extent and rapidity of steatogenesis. PLIN2 is a candidate molecular marker of sustained lipid accumulation in HepG2 cells.

Keyphrases

adipose tissue
weight gain
fatty acid
body mass index
single molecule
combination therapy