Pathological priming causes developmental gene network heterochronicity in autistic subject-derived neurons.
Simon T SchaferApua C M PaquolaShani SternDavid GosselinManching KuMonique PenaThomas J M KuretMarvin LiyanageAbed AlFatah MansourBaptiste N JaegerMaria C MarchettoChristopher K GlassJerome MertensFred H GagePublished in: Nature neuroscience (2019)
Autism spectrum disorder (ASD) is thought to emerge during early cortical development. However, the exact developmental stages and associated molecular networks that prime disease propensity are elusive. To profile early neurodevelopmental alterations in ASD with macrocephaly, we monitored subject-derived induced pluripotent stem cells (iPSCs) throughout the recapitulation of cortical development. Our analysis revealed ASD-associated changes in the maturational sequence of early neuron development, involving temporal dysregulation of specific gene networks and morphological growth acceleration. The observed changes tracked back to a pathologically primed stage in neural stem cells (NSCs), reflected by altered chromatin accessibility. Concerted over-representation of network factors in control NSCs was sufficient to trigger ASD-like features, and circumventing the NSC stage by direct conversion of ASD iPSCs into induced neurons abolished ASD-associated phenotypes. Our findings identify heterochronic dynamics of a gene network that, while established earlier in development, contributes to subsequent neurodevelopmental aberrations in ASD.
Keyphrases
- autism spectrum disorder
- attention deficit hyperactivity disorder
- intellectual disability
- induced pluripotent stem cells
- copy number
- genome wide
- spinal cord
- gene expression
- dna damage
- dna methylation
- single cell
- transcription factor
- density functional theory
- spinal cord injury
- drug induced
- working memory
- single molecule
- endothelial cells
- genome wide analysis
- finite element