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Free fatty acid receptor 4 in cardiac myocytes ameliorates ischemic cardiomyopathy.

Michael J ZhangSergey KarachenetsDylan J GybergSara PucciniChastity L HealySteven C WuGregory C ShearerTimothy D O'Connell
Published in: bioRxiv : the preprint server for biology (2024)
Our results demonstrate that Ffar4 in cardiac myocytes attenuates systolic dysfunction post-I/R, potentially by attenuating oxidative stress, preserving mitochondrial function, and modulation of cGMP signaling.
Keyphrases
  • oxidative stress
  • left ventricular
  • fatty acid
  • heart failure
  • ischemia reperfusion injury
  • blood pressure
  • nitric oxide
  • dna damage
  • mouse model
  • diabetic rats
  • induced apoptosis
  • signaling pathway