Shaymaa M M Yahya

Scopus Author ID ORCID

Publication Activity (10 Years)

Years Active: 2019-2023
Publications (10 Years): 7

Top Topics

Signaling Pathway

Oxidative Stress

Top Venues

Scientific reports

Molecular biology reports

Naunyn-Schmiedeberg's archives of pharmacology

Breast cancer research and treatment

This page only lists publications with an associated author ORCID identifier.

Publications

Heba K Nabih, Ahmad R Hamed, Shaymaa M M Yahya
Anti-proliferative effect of melatonin in human hepatoma HepG2 cells occurs mainly through cell cycle arrest and inflammation inhibition. Scientific reports 13 (1) (2023)
Heba K Nabih, Ahmad R Hamed, Shaymaa M M Yahya
Anti-proliferative effect of melatonin in human hepatoma HepG2 cells occurs mainly through cell cycle arrest and inflammation inhibition. Scientific reports 13 (1) (2023)
Ahmad R Hamed, Shaymaa M M Yahya, Heba K Nabih
Anti-drug resistance, anti-inflammation, and anti-proliferation activities mediated by melatonin in doxorubicin-resistant hepatocellular carcinoma: in vitro investigations. Naunyn-Schmiedeberg's archives of pharmacology (2023)
Heba K Nabih, Ahmad R Hamed, Shaymaa M M Yahya
Anti-proliferative effect of melatonin in human hepatoma HepG2 cells occurs mainly through cell cycle arrest and inflammation inhibition. Scientific reports 13 (1) (2023)
Shaymaa M M Yahya, Heba K Nabih, Ghada H Elsayed, Shimaa Ibrahim Abdelmenym Mohamed, Asmaa M Elfiky, Sohair M Salem
Restoring microRNA-34a overcomes acquired drug resistance and disease progression in human breast cancer cell lines via suppressing the ABCC1 gene. Breast cancer research and treatment (2023)
Heba K Nabih, Ahmad R Hamed, Shaymaa M M Yahya
Anti-proliferative effect of melatonin in human hepatoma HepG2 cells occurs mainly through cell cycle arrest and inflammation inhibition. Scientific reports 13 (1) (2023)
Shaymaa M M Yahya, Salma M Abdelnasser, Ahmad R Hamed, Osama H El Sayed, Mohsen S Asker
Newly isolated marine bacterial exopolysaccharides enhance antitumor activity in HepG2 cells via affecting key apoptotic factors and activating toll like receptors. Molecular biology reports 46 (6) (2019)