Thomas D James (Thomas James)

Publication Activity (10 Years)

Years Active: 2018-2024
Publications (10 Years): 5

Top Topics

Alcohol Use Disorder

Single Molecule

Transcranial Direct Current Stimulation

Top Venues

Research square

Synapse (New York, N.Y.)

University of Iowa

University of Iowa (PHD)

This page only lists publications with an associated author ORCID identifier.

Affiliation History

University of Iowa

United States

Postdoctoral Scholar :: Pharmacology

University of Iowa

United States

Graduate Research Assistant :: Neuroscience

Rutgers University Camden

United States

Part Time Lecturer :: Biology

Rutgers University Camden

United States

Teaching Assistant :: Biology

Education History

University of Iowa

United States

PHD :: Neuroscience

Rutgers University Center for Computational and Integrative Biology

United States

MSC

Rutgers University Camden

United States

BSC

Publications

Samantha R Pierson, Kimberly L Fiock, Ruixiang Wang, Nagalakshmi Balasubramanian, Jessica Reinhardt, Kanza M Khan, Thomas D James, Mikayla L Hunter, Benjamin J Cooper, Hannah R Williamsen, Ryan Betters, Kaancan Deniz, Gloria Lee, Georgina Aldridge, Marco M Hefti, Catherine A Marcinkiewcz
Tau pathology in the dorsal raphe may be a prodromal indicator of Alzheimer's disease. Molecular psychiatry (2024)
Catherine A Marcinkiewcz, Rui-Xiang Wang, Kanza Khan, Nagalakshmi Balasubramanian, Thomas D James, Selvakumar Pushpavathi, David Kim, Samantha Pierson, Qi Wu, Mark Niciu, Marco Hefti
Alcohol inhibits sociability via serotonin inputs to the nucleus accumbens. Research square (2023)
C Andrew Frank, Thomas D James, Martin Müller
Homeostatic control of Drosophila neuromuscular junction function. Synapse (New York, N.Y.) 74 (1) (2019)
Thomas D James, Danielle J Zwiefelhofer, C Andrew Frank
Maintenance of homeostatic plasticity at the Drosophila neuromuscular synapse requires continuous IP3-directed signaling. eLife 8 (2019)
Douglas J Brusich, Ashlyn M Spring, Thomas D James, Catherine J Yeates, Timothy H Helms, C Andrew Frank
Drosophila CaV2 channels harboring human migraine mutations cause synapse hyperexcitability that can be suppressed by inhibition of a Ca2+ store release pathway. PLoS genetics 14 (8) (2018)